What Really Causes Schizophrenia by Harold D. Foster

Author:Harold D. Foster
Language: eng
Format: mobi, pdf
Tags: Diseases, Medical, General, schizophrenia-intro, Diagnosis, Psychiatry
ISBN: 9781412015318
Publisher: Not Avail
Published: 2003-11-30T22:00:00+00:00

schizophrenia is related to COMT polymorphism. Psychiatric Genetics, 10(3), 117-124.

38. Harada, S., Tachikawa, H., and Kawanishi, Y. (2001). Glutathione S-transferase M1 gene deletion my be associated with susceptibility

to certain forms of schizophrenia. Biochemical and Biophysical Research Communications, 281, 267-271.

39. Ibid.

40. Arinami, T., Yamada, N., Yamakawa-Kobayashi, K., Hamaguchi, H., and Toru, M. (1997). Methylenetetrahydrofolate reductase variant and

schizophrenia/depression. American Journal of Medical Genetics, 74(5), 526-528.

41. Methylenetetrahydrofolate reductase: Homocysteinemia, schizophrenia, rages, depression, CNS dysfunction, neural tube defects. http://

www.kmmutants.org/07A.php

42. Miller, A.L., and Kelly, G.S. (1997). Homocysteine metabolism: Nutritional modulation and impact on health and disease. Alternative Medicine Review, 2(4), 234-254.

43. Hoffer, A. (1998) op. cit.

44. Axelrod, J. (1986). Doing research in the intramural program of the National Institutes of Health. Perspectives in Biology and Medicine, 29(3) Part 2, S131-S137.

45. Fonlupt, P., Roche, M., Cronenberger, L., and Pacheco, H. (1979).

Activation by S-adenosylhomocysteine of norepinephrine and se-

rotonin in vitro uptake in synaptosomal preparations from rat brain.

Experientia, 35(8), 1089-1090.

46. Fonlupt, P., Roche, M., Cronenberger, L., and Pacheco, H. (1979). Action of S-adenosyl-L-homocysteine on the metabolism of dopamine,

norepinephrine and serotonin in rat brain. Archives Internationales de Pharmacodynamie et de Therapie (Ghent), 240(1), 35-44.

47. Brown, N.D., Miura, G.A., Stermer-Cox, M.G., Richard, M.M., and Chiang, P.K. (1985). Unexpected increase in catecholamines in

adrenals of rats treated with 3-deazaadenosine. Biochemical and Biophysical Research Communications, 127(3), 924-928.

48. Yoshida, A., Yokogoshi, H., and Dikshit, M. (1986). Effect of methionine supplementation to a low soy protein diet on the brain free

amino acids, serotonin and catecholamines in rats. Nutritional Science Soy Protein, Japan, 7, 41-46.

49. Stoney, C.M., and Engebretson, T.O. (2000). Plasma homocysteine concentrations are positively associated with hostility and anger. Life Sciences, 66(23), 2267-2275.

134

50. Lachan et al., op. cit.

51. Novak, G., Kim, D., Seeman, P., and Tallerico, T. (2002). Schizophrenia and Nogo: Elevated mRNA in cortex, and high prevalence of a

homozygous CAA insert. Brain Research. Molecular Brain Research (Amsterdam), 107(2), 183-189.

52. Hoffer, A. (1982). The adrenochrome hypothesis of schizophrenia revisited. Journal of Orthomolecular Psychiatry, 10(2), 1-21.

53. Ibid.

54. Chamberlin, T.C. (1980). The method of multiple working hypotheses.

Science (old series), 15, 92. This is a classical geological paper which can be consulted on the Internet at http://www.accessexcellence.

com/AB/BC/chamberlin.html. Failure to follow the methodology set out by Chamberlin is the root cause of lack of progress in preventing and curing schizophrenia.

135

First they ignore you, then they laugh at you, then they fight you, then you win.

Mahatma Gandhi

136

12

PULLING THE TRIGGER

I know that most men, including those at ease with prob-

lems of the greatest complexity, can seldom accept even

the simplest and most obvious truth, if it be such as would

oblige them to admit the falsity of conclusions which they

have delighted in explaining to colleagues, which they

have proudly taught to others, and which they have

woven thread by thread into the fabric of their lives.

Leo Tolstoy (1828-1910)

Genetic inheritance plays a significant role in determining who

becomes a schizophrenic and who does not. This, of course, is

why schizophrenia tends to run in families1 and why roughly

50 percent of identical twins will eventually become a schizo-

phrenic, if their counterpart already is one.2 Nevertheless, if

genes were destiny, all such identical twins would develop the

disorder, once their sibling had been affected.

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